Diet and Kras mutations in pancreatic cancer

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Food and nutrient intakes associated with the mutational activation of the K-ras oncogene in pancreatic ductal adenocarcinoma Miquel Porta www.imim.es/URECMC/eng

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Eva Morales, Jesús Vioque, Tomàs López,Marta Crous, José Pumarega,Michelle Mendez, Núria Malats,Paco Realfor the PANKRAS II Study Group Miquel Porta www.imim.es/URECMC/eng

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Research on the causes of pancreatic cancer is a “natural meeting place” for basic science (knowledge on biological mechanisms), and epidemiology (knowledge for primary prevention).

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Research on the role of gene-environment interactions in the etiology of pancreatic cancer is generating basic knowledge on biological mechanisms, and for primary prevention.

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1 Environmental exposures. 2 Acquired genetic alterations and inherited variants. 3 Gene-gene and gene-environment interactions. Molecular epidemiology, genetics & public health Khoury M et al. Oxford University Press, 2000.

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Disease-related genetic alterations: ? Germinal or inhereted – of high penetrance ? Somatic or acquired – of low penetrance

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1 Population distribution of environmental exposures. Relative risks for exposure - disease. Interactions among exposures. Population attributable risks. Burden of illness caused by environmental exposures. Khoury M et al. Oxford University Press, 2000. Molecular epidemiology, genetics & public health 2 Population distribution of genetic alterations (acquired) and genetic variants (inherited). Burden of disease caused by genetic alterations. 3 Population distribution of gene-gene and gene-environment interactions. Clinical effects (caused by interactions). Burden of illness (caused by interactions).

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	mporta@imim.es 5 Years ago.



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